A research study has found that cerebellar inhibitory interneurons are essential for fear memory, a type of emotional memory formation. Inhibitory interneurons within the cerebellar circuitry act as gatekeepers and control the output of the cerebellar cortex. The formation of fear memory requires the activity of these interneurons. The findings may lead to a novel treatment approach for post-traumatic stress disorder.
An LSU Health New Orleans research study led by Siqiong June Liu, PhD, Professor of Cell Biology and Anatomy, has found that cerebellar inhibitory interneurons are essential for fear memory, a type of emotional memory formation. Inhibitory interneurons within the cerebellar circuitry act as gatekeepers and control the output of the cerebellar cortex. The formation of fear memory requires the activity of these interneurons.
The research team found that fear conditioning suppresses hyperpolarization-activated cyclic nucleotide-gated channels and enhances cerebellar interneuron excitability. HCN currents are similar to pacemakers in the brain because they help regulate rhythmic activity and communication between brain cells. HCN loss is driven by a learning-induced decrease in endocannabinoid levels.
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