For almost three years, scientists have raced to understand the immune responses in patients who develop severe COVID-19. — The Conversation
. We now know that in patients with severe COVID-19, many of the developing antibodies responsible for neutralizing the viral threat are simultaneously targeting their own organs and tissues. We also show that self-directed antibodies can persist for months or even years in those suffering from long COVID-19.
As researchers like us continue to study COVID-19, our understanding of the link between antiviral immunity and chronic autoimmune disease is rapidly evolving. https://www.youtube.com/embed/2ECsro9t_c0?wmode=transparent&start=0 More than three years into the pandemic, there is not one unifying explanation for why people experience the symptoms known as long COVID-19.
To prevent self-destruction, the immune system relies on an intricate series of fail-safes that are collectively termedto identify and eliminate potentially traitorous immune cells. One of the most important steps in this process occurs as the immune system builds up its arsenal against a potential threat.
When your immune system first encounters a pathogen or even a perceived threat – such as a vaccine that resembles a virus – it rapidly recruits “B” cells that have the potential to become antibody-producers. Then, any of these “naive” B cell recruits – naive being a technical term used in immunology – that demonstrate an ability to competently attack the invader are put intoHere, the cells are trained to better recognize and combat the threat.
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