Taking care of a gene during the embryonic stage can prevent autism.
Later, it was discovered that the suppressed TOP2a gene actually controls a bunch of other genes that are associated with autism in humans. All the TOP2a-controlled genes were also found to be actively binding with a protein called PRC2. So the researchers assumed that perhaps TOB2a and PRC2 together are linked to the rise of autism in a person.
In order to test this theory, they exposed another batch of zebrafish embryos to a PRC2-inhibiting drug UNC1999. Then the researchers introduced fluoroquinolones into the same embryos. The fish resulting from these embryos were found to beThis time the fish preferred to stay closer and swim together with other fish in their group. These findings suggest that the researchers were able to restore sociability in fluoroquinolones-exposed fish by subjecting them to UNC1999.
“It’s possible that these four compounds are just the tip of the iceberg in terms of substances that could be problematic for embryonic exposure,” Dr. Peterson. Therefore, further research is required to explore more possibilities and confirm the safety and effectiveness of TOP2a-inhibiting chemicals for humans.
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